Dong Gua

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Benincasa hispida (Thunb.) Cogn.

Not yet clinically reviewed

Genus: Benincasa Species: hispida Pinyin: Dong Gua
Winter melon冬瓜

Traditionally used for

  • Cough & breathing
  • Urinary & fluids
  • Mood & calm

Cautions & contraindications

  • Toxic — professional use only
Moderate evidence · 5 studies

☯ TCM Properties

Category: clearing heat
Temperature: cool
Taste: sweet, bland
Meridians: lung, large intestine, small intestine
Functions:

Promotes Urination and Reduces Edema; Clears Heat; Resolves Phlegm; Generates Fluids and Relieves Thirst; Resolves Toxicity

Traditional Chinese Uses

Dong Gua (winter melon, wax gourd) is a cool, sweet food-herb widely used in Chinese medicine to clear Heat, promote urination, and reduce edema. It benefits conditions of excess water retention, summer-heat-related thirst and irritability, and urinary difficulty. Low in calories and sodium, it is considered especially suitable for those who tend toward heat, excessive thirst, or fluid retention, and its culinary use as a cooling vegetable reflects its therapeutic properties.

Western Herbalism Properties

Actions:
diuretic

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Botanical Description

Benincasa hispida, the wax gourd or winter melon, is a vigorous annual climbing or trailing vine in the family Cucurbitaceae, believed native to southern and southeastern Asia and widely cultivated throughout tropical Asia, southern China, India, and the Caribbean for its very large edible fruit. The plant produces stout, ribbed, hispid stems several meters long bearing branched tendrils, and ascends fences and trellises or sprawls across the ground. The alternate leaves are large, palmately five- to seven-lobed, 10-25 cm across, with cordate bases and softly hairy surfaces. The plant is monoecious, bearing large solitary, bright yellow, five-lobed bell-shaped flowers 6-12 cm across, the male and female flowers on the same plant. The fruit is a massive oblong to spherical pepo 30-60 cm long, weighing up to 15-30 kg, with smooth green skin that becomes covered in a thick whitish waxy bloom at maturity, snow-white sweet flesh, and many flat, pale brown ovate seeds embedded centrally.

Active Constituents

Isomultiflorenol and isomultiflorenyl acetate

Multiflorane-type pentacyclic triterpene

Isomultiflorenol acetate is the major component of the waxy bloom on the rind of the wax gourd, which is where the plant gets its English name. It was isolated from the fruits along with new triterpenoids in a 2013 phytochemical study of the drug.

Multiflorenol

Multiflorane-type pentacyclic triterpene

Isolated from the fruits of Benincasa hispida. Multiflorenol and alnusenol have been reported among the triterpenes of this fruit that inhibit histamine release, which is the phytochemical basis usually cited for the traditional use in phlegm and allergic-type presentations.

Alnusenol

Pentacyclic triterpene

A fruit triterpene reported, with multiflorenol, to inhibit histamine release in vitro.

Stigmasterol and stigmasteryl beta-glucoside

Phytosterol and phytosterol glycoside

Phytochemical analysis of ash gourd pulp identified stigmasterol, stigmasteryl beta-glucoside and 6'-O-palmitoyl stigmasteryl beta-glucoside as the characteristic sterols of the flesh. Sterol glycosides of this kind reduce intestinal cholesterol absorption and are the likely basis of the lipid effects seen in fed-animal studies.

Cucurbitacin B, cucurbitacin E and cucurbitacin I

Cucurbitane-type triterpene

Concentration: detected qualitatively by UHPLC-Q-Orbitrap HRMS in a hydroalcoholic fruit extract; not quantified

Cucurbitacins are the intensely bitter and cytotoxic principles of the gourd family, and their detection in a concentrated hydroalcoholic extract of this fruit is worth knowing even though culinary wax gourd is bland and well tolerated. Bitterness in a cucurbit fruit signals high cucurbitacin content and the fruit should be rejected rather than dosed.

Isovitexin

C-glycosyl flavone

The characteristic C-glycosyl flavone reported from the fruit, alongside catechin, naringenin, kaempferol, quercetin and myricetin identified by LC-HRMS in a hydroalcoholic extract.

Arbutin

Hydroquinone glucoside

Isolated from the fruits of Benincasa hispida among twelve known compounds accompanying three new triterpenoids. Arbutin is hydrolysed in vivo to hydroquinone, which is the reason arbutin-containing herbs are generally not given in high dose for long periods.

⚠ Drug Interactions

Insulin, sulfonylureas, metformin and other glucose-lowering drugs

Moderate Evidence: Possible

Lyophilised ash gourd juice given to rats made prediabetic by 16 weeks of a high-fat diet significantly reduced fasting blood sugar, glucose intolerance and insulin resistance and reversed fatty liver change. A hydroalcoholic fruit extract restored glucose, triglycerides, urea and cholesterol towards normal in mice made diabetic by high-fat diet plus low-dose streptozotocin, and reduced renal fibrosis and podocyte loss. All of this is rodent data; no human glycaemic trial of the fruit has been published.

Clinical note: Ordinary culinary amounts of winter melon are unlikely to matter. If a patient is taking concentrated juice or an extract daily as therapy, treat it as a glucose-lowering agent and monitor accordingly.

Loop and thiazide diuretics

Theoretical Evidence: Theoretical

The classical indication for Dong Gua is promoting urination and reducing oedema, and diuretic activity is repeatedly claimed in the pharmacological review literature for this species. I could not verify a controlled diuretic study of the fruit against a comparator, so this remains an extrapolation from traditional use rather than a measured interaction.

Clinical note: In a patient already on a diuretic for oedema or heart failure, watch weight and, where relevant, potassium rather than assuming the herb adds nothing.

Bitter cucurbit fruit (Cucurbita or Lagenaria) mistaken for or substituted for wax gourd

Theoretical Evidence: Theoretical

Cucurbitacins B, E and I were detected by LC-HRMS in a hydroalcoholic Benincasa hispida fruit extract. Wax gourd itself is not a recognised cause of cucurbitacin poisoning, but the family it belongs to is, and bitter bottle gourd and bitter ornamental squash have caused severe vomiting and diarrhoea. The risk here is one of identification and taste, not of the authentic drug.

Clinical note: Reject any wax gourd, juice or decoction that tastes bitter, and do not press on with a preparation a patient reports as unpalatably bitter.

Evidence Tier

Moderate evidence · 5 studies

Recorded studies by study design, strongest design at the top. This is a study-design tier only, not a GRADE rating: it does not weigh risk of bias, consistency or precision.

Verified: design read from PubMed for a DOI that resolves to the cited paper Unverified: taken from the study's recorded description

Clinical Studies

Lyophilized ash gourd (Benincasa hispida (Thunb.) Cogn.) juice alleviates diet-induced prediabetes in a rat model

Athira AS, Abhijith B, Sruthi PK, Ragavamenon AC, Lankalapalli RS, Reshma MV (2025) Food & Function animal

Sprague-Dawley rats were made prediabetic with 16 weeks of a high-fat diet, showing reduced pancreatic function, insulin resistance, hyperglycaemia, dyslipidaemia and macro- and microvesicular hepatic fat. Lyophilised ash gourd juice powder then significantly reduced fasting blood sugar, glucose intolerance and insulin resistance and returned liver histology towards normal architecture. Phytochemical analysis of the pulp identified stigmasterol, stigmasteryl beta-glucoside and 6'-O-palmitoyl stigmasteryl beta-glucoside.

Hydroalcoholic extract of Benincasa hispida (Thunb.) Cogn. reinstates renal structural and functional dynamics compromised in diabetic mice: molecular insights through integration of transcriptomics and proteomics

Singh N, Kesharwani A, Gajbhiye RL, Sankar Sreenikethanam Hari H, Bharathavikru RS, Ravichandiran V, Parihar VK (2026) Journal of Ethnopharmacology animal Verified: In vitro / animal

In mice made type 2 diabetic by high-fat diet plus low-dose streptozotocin, a hydroalcoholic fruit extract restored serum glucose, triglycerides, urea and cholesterol towards normal, repaired markers of DNA damage and epigenetic disturbance in the kidney, limited podocyte loss and reduced renal inflammation and fibrosis via CD200, TGF-beta and BCl3. LC-HRMS identified 28 constituents including catechin, naringenin, kaempferol, quercetin, myricetin and cucurbitacins E, I and dihydrocucurbitacin B.

Acute and sub-chronic toxicity studies of Benincasa hispida (Thunb.) cogniaux fruit extract in rodents

Shakya A, Chaudhary SK, Bhat HR, Ghosh SK (2020) Regulatory Toxicology and Pharmacology animal Verified: In vitro / animal

A standardised 70 percent ethanolic extract of the fruit pulp produced no mortality and no behavioural signs of toxicity at the OECD limit-test dose of 2000 mg/kg in mice. In Wistar rats given 40, 200 or 1000 mg/kg daily for 90 days there were no significant changes in food and water intake, body or relative organ weights, haematology, serum biochemistry, urinalysis or the histopathology of vital organs. The authors concluded the extract is non-toxic up to 1000 mg/kg for prolonged oral use.

Gastroprotective effect of Benincasa hispida fruit extract

Rachchh MA, Jain SM (2008) Indian Journal of Pharmacology animal

Petroleum ether and methanol extracts of the fruit at 300 mg/kg orally were tested in three rodent ulcer models. In ethanol-induced gastric damage both reduced the ulcer index and vascular permeability, the methanol extract giving about 67 percent protection against 49 percent for the petroleum ether extract, compared with omeprazole 20 mg/kg. In pylorus ligation both reduced the ulcer index, with the methanol extract lowering acid output and pepsin and raising mucin markers, and in cold restraint stress both reduced ulcer index and malondialdehyde while raising catalase.

Benincasa hispida alleviates amyloid pathology by inhibition of Keap1/Nrf2-axis: Emphasis on oxidative and inflammatory stress involved in Alzheimer's disease model

Rapaka D, Bitra VR, Ummidi R, Akula A (2021) Neuropeptides animal Verified: In vitro / animal

Fruit extract at 250 and 500 mg/kg daily for 16 weeks was given to rats with aluminium chloride-induced Alzheimer-type pathology. Treated animals performed better in the Morris water maze, Y-maze and rota-rod, had restored acetylcholine, dopamine and serotonin levels, higher SOD, catalase and glutathione with lower malondialdehyde, lower TNF-alpha and IL-1beta, upregulated Keap1/Nrf2/HO-1 transcripts and less hippocampal CA3 damage. This is an Ayurvedic rather than a TCM indication for the drug and has no human support.

Historical Texts

Shen Nong Ben Cao Jing

Han dynasty
Bai Gua Zi, the seed of the white gourd, is listed in the upper class. The classical tradition therefore enters this plant through the seed rather than through the flesh, and Dong Gua Zi remains a separate drug from Dong Gua and Dong Gua Pi in modern practice.

Jin Gui Yao Lue

Han dynasty
Da Huang Mu Dan Tang, for intestinal abscess, includes melon seeds, conventionally identified as Dong Gua Ren, alongside rhubarb, moutan, peach kernel and mirabilite. This is the earliest formula context for the seed's role in draining pus from the lower burner.

Bei Ji Qian Jin Yao Fang

Tang dynasty
Sun Simiao's Wei Jing Tang, the standard formula for lung abscess, pairs Dong Gua Ren with reed stem, coix and peach kernel to expel pus and drain the phlegm-heat of the lung, the counterpart in the upper burner to the Jin Gui use in the intestine.

Ben Cao Gang Mu

Ming dynasty
Li Shizhen gives dong gua its own entry and separates the actions of the flesh, the rind and the seed, the flesh being cooling and moving water, the rind more specifically diuretic and the seed acting on pus and phlegm. Modern practice preserves this three-way split.

References

  1. Han X-n, Liu C-y, Liu Y-l, Xu Q-m, Li X-r, Yang S-l. New Triterpenoids and Other Constituents from the Fruits of Benincasa hispida (Thunb.) Cogn. . Journal of Agricultural and Food Chemistry (2013) [DOI]

This information is for educational purposes only and is not intended to replace professional medical advice. Always consult a qualified healthcare provider before using any herbal remedy, especially if you are pregnant, nursing, or taking medications.

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